Needless to say, I took it like a man and got a buzzcut, and I was pleasantly surprised to see that there were multiple new hairs growing all over my head. But to avoid similar issues in the future, I really needed something that would prevent the DHT from reaching scalp hair receptors (without reducing or blocking it systemically – . Propecia), so I started researching and that’s how I came across this article. Afterwards, I added RU58841 and a Minoxidil version which contains Azelaic Acid to my regimen and the results have been fantastic…for 3 months, or so.
The side effects of Letrozole can include those surrounding cholesterol, and this is an issue some steroid users tend to have a hard time with. The overwhelming data shows AI’s like Letrozole have very little affect on cholesterol, and if they do have any affect it’s normally insignificant. However, such data and studies rarely take into account the inclusion of anabolic steroids, specifically aromatizing anabolic steroids, and this changes the game. When examining the side effects of Letrozole, it’s important to understand the inclusion of anabolic steroids can change things. Many anabolic steroids have the ability to negatively affect cholesterol; the steroid in question will determine the total potential affect. Oral steroids are normally the harshest as they often significantly increase LDL cholesterol (bad cholesterol) and suppress HDL cholesterol (good cholesterol). Then we have injectable steroids , the their affects on cholesterol can also vary greatly. Trenbolone can have a strong, negative impact on both HDL and LDL. Testosterone can also have an impact on HDL, but normally not very strong. Hormones like Nandrolone will typically fall somewhere in the middle. With the above information in play, although it cannot significantly affect cholesterol on its own, what happens when Letrozole is used with anabolic steroids, specifically aromatizing steroids? Data shows when testosterone is used in therapeutic doses the relative affect on cholesterol is statistically insignificant. When doses are increased to supraphysiological levels, HDL suppression may occur but should not be at a true level of concern. The same data shows things, however, change greatly when the same testosterone is used with an AI like Letrozole. Data shows that a 20-25% reduction in HDL cholesterol is very possible with therapeutic or supraphysiological doses of testosterone. For this reason, a cholesterol friendly diet is important, one that’s rich in omega fatty acids, low in simple sugars and saturated fats and a lifestyle that promotes regular cardiovascular activity.
For me, it would be a quality-of-life question, not a performance issue. If the HGH weren't so expensive, I'd probably continue with it, at least until I had a good reason not to, like some new evidence that it makes you grow extra ears. (The side effects of HGH are reportedly mild—one is fluid retention.) If nothing else, it helped my eyesight, and I had more energy. Lately, I've been reading studies about how endurance athletes suffer from low testosterone, which leads to early signs of osteoporosis, so I'm going to continue to monitor my levels and, if they drop too far, consider boosting them with the cream.
With the EPO, even if somebody gave it away, I wouldn't go down that road. Using it is too much of a literal and figurative headache, and if you get sloppy there's always the danger of nasty results. And I would never touch steroids again, unless I had some specific medical need. It's all just too powerful, too strange, and it's hard to read a list of the side effects and not feel like you're playing Russian roulette.
As for the larger issue of drugs in sports, eight months in the world of the artificially enhanced convinced me more than ever that it's critical for an organization like the World Anti-Doping Agency to succeed. This group, founded after the Salt Lake Olympics by Canadian anti-doping leader Dick Pound, represents the most serious international attempt to come to grips with sports doping. WADA is the logical response to an argument that gets aired from time to time: that since cheating is impossible to eliminate, the only recourse is to simply legalize everything—that way, no athlete has a hidden advantage over another, since everyone would be free to try anything that might increase endurance.
Like a lot of powerfully bad ideas, that one has a certain mad logic. But it would turn every sport into a test of how much damage an athlete was willing to risk to improve performance, and would basically force every serious athlete to cheat and risk his or her health. Athletic contests would have a strange life-or-death quality. If we don't keep drugs out of these events, they become freak shows, the athletes like gladiators—with us playing the role of decadent Romans, urging them on.
Besides, on a fundamental level, drugs ruin the simple joy of competition. With drugs in the mix, it's not about the athletes, it's about the chemistry.
Now that I was off the program, I started to think about what I'd train for next. Probably something shorter than the PBP—say, the Canadian Ski Marathon, a two-day, 100-mile event. I got a calendar out and began to work on the training schedule. I'd done the race before and knew it would be long, cold, and brutal.
Sounded fun to me. And this time I'd do it on my own.